Plasma renin activity in psychosocial hypertension of CBA mice.
نویسندگان
چکیده
30. Smith OA Jr, Rushmer RF, Lasher EP: Similarity of cardiovascular responses to exercise and to diencephalic stimulation. Am J Physiol 198: 1139-1142, 1960 31. Ross, J Jr: Afterload mismatch and preload reserve: A conceptual framework for the analysis of ventricular function. Prog Cardiovasc Dis 18:~255-264, 1976 32. Vikhert AM, Cherpachenko NM: Changes in metabolism of undamaged sections of myocardium following infarction. Circ Res 35: 182— 191,1974 33. Wyatt HL, Forrester JS, daLuz PL, Diamond GA, Ghagrasulis R, Swan HJC: Functional abnormalities in nonoccluded regions of myocardium after experimental coronary occlusion. Am J Cardiol 37: 366-372, 1976 34. Tyberg JV, Forrester JS, Wyatt HL, Goldner SJ, Parmley WW, Swann HJC: An analysis of segmental ischemic dysfunction utilizing the pressure-length loop. Circulation 49: 748-754, 1974 35. Master AM, Rosenfeld I: Two-step exercise test: Current status after twenty-five years. Mod Concepts Cardiovasc Dis 36: 19-24, 1967 36. Zaret BL, Strauss HW, Herley PJ, Natarajan TK, Pitt B: A noninvasive scintiphotographic method for detecting regional ventricular dysfunction in man. N Engl J Med 284: 1165-1170, 1971 37. Nieminen M, Heikkila J: Echoventriculography in acute myocardial infarction IV. Clinical correlations and implication of the non-infarcted myocardium. Am J Cardiol 38: 1-8, 1976 38. Bruce RA, Cobb LA, Katamura S. Morledge JH, Andrews WW, Fuller TJ: Exertional hypotension in cardiac patients. Circulation 19: 543-551, 1959 39. Thomson PD, Keleman MH: Hypotension accompanying the onset of exertional angina. A sign of severe compromise of left ventricular blood supply. Circulation 52: 28-32, 1975 40. Mean WF, Pyter HR, Trombold JC, Frederick RC: Successful resuscitation of two near simultaneous cases of cardiac arrest with a review of fifteen cases occurring during supervised exercise. Circulation 53: 187-189,1976
منابع مشابه
Increased vascular sensitivity to angiotensin ii in psychosocial hypertensive mice.
CBA mice develop hypertension when placed in complex population cages that facilitate social interactions and competition for territory. After 1 month, these mice have normal plasma renin levels, but blockade of converting enzyme lowers blood pressure to normal. To test the possibility that this normal-renin hypertension is caused by enhanced pressor responsiveness to angiotensin II (AII), we e...
متن کاملThe effect of aspirin therapy on the plasma renin activity in myocardial infarction
In this study, the changes of renin system activity and the effects of aspirin therapy on the plasma renin activity ( PRA) in myocardial infarction were investigated. Fifty nine patients with myocardial infarction were selected. By suppressed in the former group which was statistically significant ( p<0.01).
متن کاملHypertensive response to acute stress is attenuated in interleukin-6 knockout mice.
This study tested the hypothesis that the inflammatory cytokine, interleukin-6, contributes to the hypertensive response to acute psychosocial stress, caused by switching male mice to a cage previously occupied by a different male mouse. Male C57BL6 (WT) and interleukin-6 (IL-6) knockout (KO) mice were implanted with biotelemetry devices to monitor mean arterial pressure, heart rate, and motor ...
متن کاملAPPARENT MINERALOCORTICOID EXCESS IN THREE SIBLINGS
Three siblings (1 boy, 2 girls) with hypertension and hypokalemia are presented, two with low plasma aldosterone and suppressed renin activity and the eldest with a high renin and aldosterone level due to secondary changes in her kidneys. Urinary tetrahydrocortisol (THF) was increased relative to tetrahydrocortisone (THE). Cortisol ring A reduction constant was also lower than normal. Thes...
متن کاملIncreased vasodilator responses to acetylcholine in psychosocial hypertensive mice.
Responsiveness to endothelium-dependent (acetylcholine and A23187) and endothelium-independent (nitroprusside and 8-bromo cyclic guanosine 3',5'-monophosphate [cGMP]) vasodilators was examined in two vascular preparations from hypertensive and normotensive mice. CBA Agouti mice were made hypertensive by exposure to social stress in a complex population cage. After 2 months, the hindquarter vasc...
متن کاملDecreased susceptibility to renovascular hypertension in mice lacking the prostaglandin I2 receptor IP.
Persistent reduction of renal perfusion pressure induces renovascular hypertension by activating the renin-angiotensin-aldosterone system; however, the sensing mechanism remains elusive. Here we investigated the role of PGI2 in renovascular hypertension in vivo, employing mice lacking the PGI2 receptor (IP-/- mice). In WT mice with a two-kidney, one-clip model of renovascular hypertension, the ...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
عنوان ژورنال:
- Circulation research
دوره 42 4 شماره
صفحات -
تاریخ انتشار 1978